IJMS, Free Full-Text
Por um escritor misterioso
Last updated 26 junho 2024
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Multiple sclerosis (MS) is a chronic neuroinflammatory disease of the central nervous system (CNS) affecting nearly three million humans worldwide. In MS, cells of an auto-reactive immune system invade the brain and cause neuroinflammation. Neuroinflammation triggers a complex, multi-faceted harmful process not only in the white matter but also in the grey matter of the brain. In the grey matter, neuroinflammation causes synapse dysfunctions. Synapse dysfunctions in MS occur early and independent from white matter demyelination and are likely correlates of cognitive and mental symptoms in MS. Disturbed synapse/glia interactions and elevated neuroinflammatory signals play a central role. Glutamatergic excitotoxic synapse damage emerges as a major mechanism. We review synapse/glia communication under normal conditions and summarize how this communication becomes malfunctional during neuroinflammation in MS. We discuss mechanisms of how disturbed glia/synapse communication can lead to synapse dysfunctions, signaling dysbalance, and neurodegeneration in MS.
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IJMS, Free Full-Text
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IJMS Free Full Text Recent Insights From Molecular Dynamics Simulations For G Protein 84405
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IJMS, Free Full-Text
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Ijms Free Full Text Delayed Onset Of Age Dependent Changes In Ultrastructure Of Myocardial 116100
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IJMS, Free Full-Text
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IJMS Free Full-Text Therapeutic Potential Of Repeated, 59% OFF
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IJMS, Free Full-Text
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IJMS Free Full-Text Research Advances In Mechanical, 52% OFF
Ijms Free Full Text The Effect Of The Chorion On Size
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IJMS Free Full Text A High Molecular Mass 26650
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